andersenpa Posted September 8, 2006 Share Posted September 8, 2006 What lives there..... chest wall - bone and musculature vertebral bodies intercostal nerves parietal and visceral pleura lung pericardium myocardium aorta esophagus diaphragm upper abdominal structures- liver, GB, stomach, transverse colon, spleen, pancreas ................. Quote Link to comment Share on other sites More sharing options...
JenGintheED Posted September 8, 2006 Share Posted September 8, 2006 If the ddx is simply CP, 10 isn't hard..... but 10 menstrual-related causes of CP? Ectopic endometrium, blood-loss anemia, psychogenic secondary to PMS (hate going there, though)... that's all that comes to mind. Others (adding to doboy's list): 6. pericarditis 7. pleurisy 8. pneumonia 9. PE 10. CVD-related vasculitis, such as SLE 11. MVP 12. costochondritis 13. muscle strain (diaphragm, intercostals, chest wall) 14. traumatic 15. esophageal spasm 16. esophageal rupture (Boerhaave) vs. Mallory-Weiss 17. esophagitis (fungal like Candida, viral like HSV) 18. Hiatal hernia 19. zoster 20. pulmonary HTN 21. cardiomyopathy CP ddx is HUGE :D Quote Link to comment Share on other sites More sharing options...
andersenpa Posted September 12, 2006 Share Posted September 12, 2006 As always, LA is all over it. Although bethp hit on what I was suggesting. Ectopic emdometrial tissue in the thoracic cavity leading to PTX. (but we all know you cheated by looking it up! :rolleyes:) Doboy- you say "TAA", does the presence of a thoracic aortic aneurysm cause pain? Quote Link to comment Share on other sites More sharing options...
Guest lisnek Posted September 12, 2006 Share Posted September 12, 2006 Doboy- you say "TAA", does the presence of a thoracic aortic aneurysm cause pain? i assume if it were dissecting/rupturing it would cause pain?? Quote Link to comment Share on other sites More sharing options...
JenGintheED Posted September 24, 2006 Share Posted September 24, 2006 From another thread... I am working with a very very very smart doctor, we were discussing asthma exacerbation associated with URI, specifically viral influenced asthma attacks. She asked me what we can do to prophylax (protect) the patient from having an asthma attack from this viral upper respiratory infection (common head cold) and I discussed various medicines which stabilize the inflammatory response that causes an asthma attack. She shot down every one of my ideas, and then told me Viruses don't cause the same asthma response as allergens, they effect a different route to cause bronchospasm.....so the medications would be ineffective. It's a great thread and I don't want to hijack it by going off on this tangent... but as is consistent with (one of) my weakness(es), I've gotta know. Unfortunately I have knees, shoulders, imms schedules and lots more to be reading about before getting back to clinicals tomorrow. So does anyone know HOW a respiratory virus provokes bronchospasm? Quote Link to comment Share on other sites More sharing options...
kargiver Posted September 24, 2006 Share Posted September 24, 2006 From another thread... It's a great thread and I don't want to hijack it by going off on this tangent... but as is consistent with (one of) my weakness(es), I've gotta know. Unfortunately I have knees, shoulders, imms schedules and lots more to be reading about before getting back to clinicals tomorrow. So does anyone know HOW a respiratory virus provokes bronchospasm? LA, the question you are asking has many, many, many answers. There is t-cell mediated immunopathology, recruitment of neutrophils and macrophages to the virus-infected cells leading to cellular damage, release of IL-11, IL-8, and other chemo/cytokines that contribute to localized inflammation, and other scenarios that contribute to localized (and on a macroscopic level) inflammation. Further, there is evidence of viruses interfering with acetylcholine regulation leading to bronchoconstriction. But as a guiding principle, viruses usually cause asthma by the infammatory response (non-IgE mediated) aimed at them in the lung (some recent literature suggests a significant role for NK cells which are not responsive to corticosteroids), not by the common underlying pathology (IgE mediated) commonly known as "asthma." G PS - now you got me reading asthma articles... :) Quote Link to comment Share on other sites More sharing options...
andersenpa Posted November 2, 2006 Share Posted November 2, 2006 Which dermatome crosses ther nipple line? The thumb? The groin? Quote Link to comment Share on other sites More sharing options...
doboy Posted November 3, 2006 Share Posted November 3, 2006 Nipples- T4 Thumb- C6 S2, 3, 4 keep the penis off the floor.... Quote Link to comment Share on other sites More sharing options...
andersenpa Posted November 4, 2006 Share Posted November 4, 2006 Doboy- groin, not genitals......L1 http://mywebpages.comcast.net/epollak/PSY255_pix/dermatomes-netter2.JPG Quote Link to comment Share on other sites More sharing options...
andersenpa Posted November 4, 2006 Share Posted November 4, 2006 Next, easy- what are the common "reversal agents" for: Narcotics Benzodiazepines Heparin Coumadin Calcium Channel Blockers Beta Blockers Neuromuscular Blockers Quote Link to comment Share on other sites More sharing options...
plouffes Posted November 4, 2006 Share Posted November 4, 2006 Next, easy- what are the common "reversal agents" for: Narcotics Benzodiazepines Heparin Coumadin Calcium Channel Blockers Beta Blockers Neuromuscular Blockers let's see..I'll take a shot at it.. Narcan, Flumazinil, Protamine?, Vit. K, Calcium Chloride, Neo-Synephrine?, Neostigmine Quote Link to comment Share on other sites More sharing options...
kargiver Posted November 4, 2006 Share Posted November 4, 2006 for the beta blockers... Glucagon Quote Link to comment Share on other sites More sharing options...
andersenpa Posted November 13, 2006 Share Posted November 13, 2006 This came up through another thread.... what are the clinical features of the neuroleptic malignant syndrome? And for bonus, how is it clinically different from malignant hyperthermia? Quote Link to comment Share on other sites More sharing options...
Guest leavinthelab Posted November 14, 2006 Share Posted November 14, 2006 I'll stop lurking and take a stab... Neuroleptic malignant syndrome is characterized by fever, rigidity and cognitive changes. It is reported to occur in people taking central dopaminergic drugs and occurs within the first few days to weeks of starting the meds. Is it differentiated from malignant hyperthermia by onset and drug exposure? (Since malignant hyperthermia occurs within an hour or so following administration of anesthetic meds) Quote Link to comment Share on other sites More sharing options...
Guest pac4hire Posted November 20, 2006 Share Posted November 20, 2006 My turn... What is the difference between SIRS, Sepsis, Sever Sepsis and Septic Shock? Quote Link to comment Share on other sites More sharing options...
andersenpa Posted November 20, 2006 Share Posted November 20, 2006 I'll stop lurking and take a stab...Neuroleptic malignant syndrome is characterized by fever, rigidity and cognitive changes. It is reported to occur in people taking central dopaminergic drugs and occurs within the first few days to weeks of starting the meds. Is it differentiated from malignant hyperthermia by onset and drug exposure? (Since malignant hyperthermia occurs within an hour or so following administration of anesthetic meds) Nice, leavin. The main difference I was thinking of is the setting in which it occurs- as you mentioned, after inhalational agents-OR, ICU, etc... Quote Link to comment Share on other sites More sharing options...
pwauburn Posted November 23, 2006 Share Posted November 23, 2006 Some things that I needed to know for ER rotation... How do you treat a patient with SVT (supraventricular tachy) that is hemodynamically stable? What if they are hemodynamically unstable with SVT? Name as many causes of PEA (pulseless electrical activity) as you can... First thing(s) to administer to a patient with PEA? With what ratio of lidocaoine/bicarb do you buffer lidocaine? In what parts of the body do you not use lidocaine with epinephrine? X-ray sign that is seen with a kid that has epiglottitis? X-ray sign that is seen with viral croup? X-ray sign that is seen in a patient that has a fracture to the proximal end of the radius (not talking about anterior fat pad)? Invasive and non-invasive immediate (temporary) treatments for a patient that has PEA due to cardiac tamponade? Quote Link to comment Share on other sites More sharing options...
Guest lisnek Posted November 23, 2006 Share Posted November 23, 2006 what are (a few) causes for equal and bilat: dilated pupils fixed/pinpoint pupils (hint-think drugs) Quote Link to comment Share on other sites More sharing options...
andersenpa Posted November 28, 2006 Share Posted November 28, 2006 What is white clot syndrome? Quote Link to comment Share on other sites More sharing options...
Azgal Posted November 28, 2006 Share Posted November 28, 2006 Not sure if you have received any responses, but here is what I found on a quick search. White Clot Syndrome: It has been reported that patients on heparin sodium may develop new thrombus formation in association with thrombocytopenia, resulting from irreversible aggregation of platelets induced by heparin the so-called ''white clot syndrome.'' The process may lead to severe thromboembolic complications like skin necrosis, gangrene of the extremities that may lead to amputation, myocardial infarction, pulmonary embolism, stroke and possibly death. There, heparin sodium administration should promptly be discontinued if a patient develops new thrombosis in association with thrombocytopenia. Quote Link to comment Share on other sites More sharing options...
Azgal Posted November 28, 2006 Share Posted November 28, 2006 I did a quick search and this is what I found, hope it helps.:D White Clot Syndrome: It has been reported that patients on heparin sodium may develop new thrombus formation in association with thrombocytopenia, resulting from irreversible aggregation of platelets induced by heparin the so-called ''white clot syndrome.'' The process may lead to severe thromboembolic complications like skin necrosis, gangrene of the extremities that may lead to amputation, myocardial infarction, pulmonary embolism, stroke and possibly death. There, heparin sodium administration should promptly be discontinued if a patient develops new thrombosis in association with thrombocytopenia. Quote Link to comment Share on other sites More sharing options...
andersenpa Posted November 29, 2006 Share Posted November 29, 2006 I did a quick search and this is what I found, hope it helps.:D White Clot Syndrome: It has been reported that patients on heparin sodium may develop new thrombus formation in association with thrombocytopenia, resulting from irreversible aggregation of platelets induced by heparin the so-called ''white clot syndrome.'' The process may lead to severe thromboembolic complications like skin necrosis, gangrene of the extremities that may lead to amputation, myocardial infarction, pulmonary embolism, stroke and possibly death. There, heparin sodium administration should promptly be discontinued if a patient develops new thrombosis in association with thrombocytopenia. :rolleyes: This is a "pimping" session thread, so it's more for posing a question to test the knowledge of students out there on the forum- not for my own knowledge.... (ie I wouldn't ask the question unless I knew the answer!) thanks though :rolleyes: :rolleyes: Quote Link to comment Share on other sites More sharing options...
Guest RillaK Posted November 29, 2006 Share Posted November 29, 2006 I'm about to take boards, and you guys are making me want to kill myself. :) Quote Link to comment Share on other sites More sharing options...
MadelinePAC Posted November 30, 2006 Share Posted November 30, 2006 Please don't, white clot syndrom is not on the boards I promise Quote Link to comment Share on other sites More sharing options...
Guest lisnek Posted December 12, 2006 Share Posted December 12, 2006 for the PA students: what are the 5 w's of post-op fever? 1. 2. 3. 4. 5. what does ****T stand for? (common causes of hematuria...) S- H- I- T- T- Quote Link to comment Share on other sites More sharing options...
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