JustaPA Posted November 16, 2004 Share Posted November 16, 2004 Here is a question:how does one permanently rid a patient of athlete's foot? amputation Quote Link to comment Share on other sites More sharing options...
Marlene G Posted November 16, 2004 Share Posted November 16, 2004 You guys are on the mark. It is my right foot - it has been there for 26 years, now and it just won't quit. From what I have heard - up to 75% of cases cannot be cured. I have some topical meds I am using and fear going for orals - considering hepatic issues. Quote Link to comment Share on other sites More sharing options...
JenGintheED Posted November 16, 2004 Share Posted November 16, 2004 What are some of the findings on examination of a patient with a peritonsillar abscess? Quote Link to comment Share on other sites More sharing options...
pahopeful Posted November 16, 2004 Author Share Posted November 16, 2004 What are some of the findings on examination of a patient with a peritonsillar abscess? hot potato voice drooling displacement of the uvula contralateral to the involved tonsil possibly enlarged, erythematous, or exudative tonsil(s) there are many more, but those are some of the basics Quote Link to comment Share on other sites More sharing options...
Guest neuroracer Posted November 16, 2004 Share Posted November 16, 2004 What is a hot potato voice? Quote Link to comment Share on other sites More sharing options...
Guest jcapers Posted November 16, 2004 Share Posted November 16, 2004 Major complication seen in a patient with portal htn/ascites is SBP.Not to mention hepatic renal syndrome,hemorrhage,metabolic changes(hypoglycemia,hypokalemiahyponatremia, and hypophosphatemia)Oh,I forgot to mention cadiovascular changes due to hypovolemia which in turn decreases cardic output.One more,encephalopathy.Pahopeful,I hope this helps your inquiry.By the way ,I too am a PA-S at UNTHSC. I'm in the 2005 class and my b-day is also on 02-14.Its a small world. Quote Link to comment Share on other sites More sharing options...
SilverPA Posted November 16, 2004 Share Posted November 16, 2004 Okay, we all know infection is a cause of fever. Name 5 other causes. While you're at it, hyperpyrexia refers to elevation above ? degrees C and F and hypothermia is ? (rectal temps). Quote Link to comment Share on other sites More sharing options...
JenGintheED Posted November 16, 2004 Share Posted November 16, 2004 What is a hot potato voice? Bake a potato. While it is steaming hot, stuff your mouth - then try to talk! That's a "hot potato" voice. Hopeful - right on. Also fever, tachycardia, trismus & cervical adenopathy. I'm so happy to see this thread come alive again! Quote Link to comment Share on other sites More sharing options...
JenGintheED Posted November 16, 2004 Share Posted November 16, 2004 Okay, we all know infection is a cause of fever. Name 5 other causes. While you're at it, hyperpyrexia refers to elevation above ? degrees C and F and hypothermia is ? (rectal temps). Malignant hyperthermia - reaction between succinylcholine & halothane. Environmental/exposure... Quote Link to comment Share on other sites More sharing options...
pahopeful Posted November 16, 2004 Author Share Posted November 16, 2004 Okay, we all know infection is a cause of fever. Name 5 other causes. While you're at it, hyperpyrexia refers to elevation above ? degrees C and F and hypothermia is ? (rectal temps). Cancer of all kinds can cause fever as well as rarer things like tumor lysis syndrome. Speaking of tumor lysis syndrome, anybody know the signs/symptoms and conditions associated with it?? Quote Link to comment Share on other sites More sharing options...
pahopeful Posted November 16, 2004 Author Share Posted November 16, 2004 Okay, we all know infection is a cause of fever. Name 5 other causes. While you're at it, hyperpyrexia refers to elevation above ? degrees C and F and hypothermia is ? (rectal temps). hyperpyrexia >106 degrees F mild hypothermia 32-35 degrees C moderate hypothermia 29-32 degrees C severe hypothermia <29 degrees C I'm not sure if these are rectals but I do know that they are based on measurements of core temperature which can be measured a couple of different ways (bladder, rectum, esophageal). Am I on the right track? Quote Link to comment Share on other sites More sharing options...
merseur Posted November 16, 2004 Share Posted November 16, 2004 Five causes of fever: W'S5 Wind-Atelectasis Water-UTI Walking-DVT wonder drugs wound infection-abscess Hypothermia-<35 C Hyperthermia->41 C Quote Link to comment Share on other sites More sharing options...
SilverPA Posted November 16, 2004 Share Posted November 16, 2004 I like everyone's response. The temps are great as are the causes of fever. Some not mentioned: trauma, some blood disorders (acute hemolytic anemia), and some immune disorders (collagen vascular disease) per Bates'. Quote Link to comment Share on other sites More sharing options...
JenGintheED Posted November 19, 2004 Share Posted November 19, 2004 A patient presents to the ED with recent onset of a painless descending paralysis with no sensory deficits and no alteration of consciousness. No gastrointestinal symptoms are reported. What entity does this presentation strongly suggest? (from emedhome question-a-day) Quote Link to comment Share on other sites More sharing options...
v_chicky Posted November 19, 2004 Share Posted November 19, 2004 i think i finally know one! hot potato voice = peritonsilar abscess/cellulitis. oh and angel, shouldn't you be studying for pharm?! :p Quote Link to comment Share on other sites More sharing options...
Marlene G Posted November 19, 2004 Share Posted November 19, 2004 I think I am a fool because I am thinking that I should know these off the top of my head, but a little birdy tells me the PA-S people are looking them up. LOL! Quote Link to comment Share on other sites More sharing options...
merseur Posted November 20, 2004 Share Posted November 20, 2004 I am stump!! Quote Link to comment Share on other sites More sharing options...
JenGintheED Posted November 20, 2004 Share Posted November 20, 2004 I think I am a fool because I am thinking that I should know these off the top of my head, but a little birdy tells me the PA-S people are looking them up.LOL! If you knew all these, then you'd miss out on the fun of learning :p And that's what we're here for, right? This one stumped me too, when I first saw it, but I posted it once I saw the answer because we all learned it in micro (hint). Quote Link to comment Share on other sites More sharing options...
merseur Posted November 20, 2004 Share Posted November 20, 2004 Is it BOTULISM? Quote Link to comment Share on other sites More sharing options...
JenGintheED Posted November 20, 2004 Share Posted November 20, 2004 c'est Botulism! "The classic feature of botulism is a descending, painless symmetric paralysis. There may or may not be accompanying symptoms of gastroenteritis (Rosen’s Emergency Medicine, 5th. Edition, pg. 1524). " This threw me for a loop, because I associate GI sx with food poisoning. I did a little exploring, and found a LOT of contradictory info. Here's what I found - 1. What all the sources have in common is that botulism presents as a descending, symmetrical paralysis, beginning with cranial nerve involvement - diplopia, fixed/dilated pupils, dysphagia, xerostomia. Sensory function and mental status are normal, though there may be agitation. It is the descending/symmetrical part that separates botulism from other paralyses. 2. There is a lot of contradictory info out there on where or not n/v/d is the first presenting sx. Rosen's and Harrison's both say that the GI sx may present before OR after the paralytic sxs. Goldblaum's Toxicology and Current say that GI sxs come first. 3. Some sources say that food-borne botulism always occurs with groups of people, but other sources report food-borne botulism occuring in some individuals and not in others who ate the same food. The sources that report botulism in individuals have documented cases presented, and the sources that report groups only did not have sources. 4. The studies that looked into individual cases of botulsim also reported that every case was initially misdiagnosed in the ER. 5. Wound botulism, not the gangrenous kind, is being found in IVDAs. (Rare presentation, but new info to me). 6. Infant botulism is only associated with honey in less than 20% of the cases. Medicine is so cool (and I'm such a nerd)... :p Quote Link to comment Share on other sites More sharing options...
pahopeful Posted November 21, 2004 Author Share Posted November 21, 2004 I think I am a fool because I am thinking that I should know these off the top of my head, but a little birdy tells me the PA-S people are looking them up.LOL! Aw Marlene...if I can even think of one possible differential for some of these I'm proud! Usually I'm into the electronic reference books lookin em up...that's not cheating by the way (and I would know, I started this thread..haha!) pahopeful Quote Link to comment Share on other sites More sharing options...
Marlene G Posted November 21, 2004 Share Posted November 21, 2004 Thanks. "Thumbs up." The electronic references are great. :) Quote Link to comment Share on other sites More sharing options...
JenGintheED Posted December 20, 2004 Share Posted December 20, 2004 Courtesy of NCEMI question of the day... "After Alice touched the March Hare, she might have developed an ulcer on her hand and regional adenopathy. What is the likely diagnosis, and what is considered the treatment of choice? " Quote Link to comment Share on other sites More sharing options...
merseur Posted December 20, 2004 Share Posted December 20, 2004 Is this the dz that most wool workers get? I forgot the name. Its been a year since Infectious Dz. Quote Link to comment Share on other sites More sharing options...
Guest neuroracer Posted December 20, 2004 Share Posted December 20, 2004 Is that tularemia? I have no idea, I saw something about a rabbit. ;) Quote Link to comment Share on other sites More sharing options...
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