Jump to content

Case No. 3 - Edison Medicine


Recommended Posts

I just wanted to comment that you have a great looking web site. Being a headache guy I rarely seek ECGs anymore (except for studying for the recerts). So, without embarrassing myself too much, do I see delta waves? Then I would suggest WPW and an ablation of the extra pathway would hopefully be curative. But I will wait to hear from the real brains here. But again, a great looking website. I would love to do the same for the headache world.

Link to comment
Share on other sites

Guest JMPA
Hi All,

 

Case no. 3 is posted over at my EM education site - take a look and let me know what you think. This is a great learning case... cards in a youngin. Looking forward to your comments.

 

http://www.empaguru.com/?p=67

 

E, take a look. You'll love it.

 

G

 

V tach until proven otherwise.

Link to comment
Share on other sites

Gracias about the site comments - most appreciated.

 

Good question - delta waves? Hard to say at a rate of 230 bpm, and with the history of transposition of great vessels, who knows what is in there for scar tissue. But JMPA is right - gotta treat it as V-tach until proven otherwise.

 

I'll reveal more as it develops with responses.

 

G

Link to comment
Share on other sites

Guest JMPA
Gracias about the site comments - most appreciated.

 

Good question - delta waves? Hard to say at a rate of 230 bpm, and with the history of transposition of great vessels, who knows what is in there for scar tissue. But JMPA is right - gotta treat it as V-tach until proven otherwise.

 

I'll reveal more as it develops with responses.

 

G

 

failure of adenosine would point away from svt in a wide comlex tachycardia, no long acting blocker should be used, the next DOC would be amiodarone if stable otherwise defibrillate

Link to comment
Share on other sites

Gracias about the site comments - most appreciated.

 

Good question - delta waves? Hard to say at a rate of 230 bpm, and with the history of transposition of great vessels, who knows what is in there for scar tissue. But JMPA is right - gotta treat it as V-tach until proven otherwise.

 

I'll reveal more as it develops with responses.

 

G

 

Sorry, I thought the V-tach was a given. I thought you were looking for the cause of the V-tach and with his history of congenital deformities and, this is where I'm not that skilled, I thought I saw delta waves in his post-conversion strip, that this could be WPW that precipitated V-tach.

Link to comment
Share on other sites

Always correct to assume that in the presence of a wide-complex tachycardia that it is V-Tach until proven otherwise. However, and this is where it gets sticky, what if it is WPW (SVT with aberrancy)? That has been a fair amount of literature about this in the EM world lately, and procainamide has definitely made a comeback. I have used it several times myself to terminate SVTs with aberrancy or WPW cases.

 

JMJ, i agree, there is some notching that is concerning but after the fact, it wasn't felt to be WPW (by peds cards at the local tertiary center). They thought is was something else.

 

There is something that everyone is overlooking though - take a look again at the history - what is missing?

 

G

Link to comment
Share on other sites

 

 

There is something that everyone is overlooking though - take a look again at the history - what is missing?

 

G

 

Looks like borderline LVH. If I recall, those pts (esp if it's HCM) are more likely to break into an arrhythmia after blunt trauma, such as falling off a skateboard. With a h/o of transposition, I doubt this particular pt has HCM, but perhaps the hypertrophy applies.

Link to comment
Share on other sites

Was the surgery completely corrective as a child? If not, I'd venture if his vessels are transposed the vtach could be secondary to hypoxia/cardiac ischemia. For an otherwise healthy kid his sats (though I hate pulse ox) are borderline toilet. The picture you painted seems also seems to rule out a pnuemo, but what are the over all lung sounds? Especially given hx of asthma.

 

If truly cardiac, probably consider an amiodarone drip considering he's stable, and you'd get the both the artia/ventricles. If all else fails, sync cardiovert that sucker up. Monitoring this thread for the answer.

Link to comment
Share on other sites

Guest JMPA
Always correct to assume that in the presence of a wide-complex tachycardia that it is V-Tach until proven otherwise. However, and this is where it gets sticky, what if it is WPW (SVT with aberrancy)? That has been a fair amount of literature about this in the EM world lately, and procainamide has definitely made a comeback. I have used it several times myself to terminate SVTs with aberrancy or WPW cases.

 

JMJ, i agree, there is some notching that is concerning but after the fact, it wasn't felt to be WPW (by peds cards at the local tertiary center). They thought is was something else.

 

There is something that everyone is overlooking though - take a look again at the history - what is missing?

 

G

neurogenic cardiac injury seems to fit the history.

Link to comment
Share on other sites

neurogenic cardiac injury seems to fit the history.

 

Definitely does - his CT scan of his head and neck were negative. That crossed my mind while treating him as well...

 

I'll cut to the chase - I thought there would be more interest in a V-Tach in a 13 yo patient after a fall. This is a not-so-classic presentation of commotio cordis, The patient hasn't died from their arrhythmia yet. Depending on where you hit your chest can precipitate different rhythms (for example, hitting your sternum directly puts you at a higher risk for VF whereas hitting your lower chest wall puts you at risk for VT). Complicating the picture was the prior history of transposition repair - but the last cath done on patient the year before showed no underlying pathology. The peds cardiologists believed ultimately though it was the fall and landing on his chest that caused the initial arrhythmia.

 

Ultimately, the patient was treated with amiodarone and vomited during administration. His BP began to drop, his LOC started decreasing so a single 50J DC cardioversion fixed the problem. Once cardioverted, patient was and remained asymptomatic. He was watched in the PICU for 24 hours and D/C to home from there.

 

About 35% of Commotio Cordis cases are successfully resuscitated, but those stats only are of those reported cases and of those cases, nearly all presented in VF. The take home point I want to make is that it can also present as VT, polymorphic VT, VF, or any SVT. There have even been reported cases of heart block occurring.

 

VT and VF are pretty straight forward to deal with - but add in the age and the sphincter factor tends to climb for all in the room. We walked this patient and the family through everything we did and it helps to make all of the difference.

 

G

Link to comment
Share on other sites

Archived

This topic is now archived and is closed to further replies.

×
×
  • Create New...

Important Information

Welcome to the Physician Assistant Forum! This website uses cookies to ensure you get the best experience on our website. Learn More